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CYP2C19 Genotype, Clopidogrel Metabolism, Platelet Function, and Cardiovascular Events, December 28, 2011, Holmes et al. 306 (24): 2704 � JAMA Genetic Epidemiology Group, Department of Epidemiology and Public Health, University College London, London, United Kingdom (Drs Holmes, Shah, Hingorani, and Casas); Faculty of Epidemiology and Public Health, London School of Hygiene and Tropical Medicine, London (Drs Perel and Casas); and Centre for Clinical Pharmacology, Division of Medicine, University College London (Drs Shah and Hingorani). Early Communication about an Ongoing Safety Review of clopidogrel bisulfate (marketed as Plavix) Quick Links: Skip to main page content Skip to Search Skip to Topics Menu Skip to Section Content Menu Skip to Common Links This information reflects FDA�s current analysis of available data concerning these drugs. Posting this information does not mean that FDA has concluded there is a cause and effect relationship between the drug products and the emerging safety issue. Nor does it mean that FDA is advising health care professionals to discontinue prescribing these products. FDA is considering, but has not reached a conclusion about whether this information warrants any regulatory action. FDA intends to update this document when additional information or analyses become available Clopidogrel Reviews Ratings at Drugs.com A B C D E F G H I J K L M N O P Q R S T U V W X Y Z Advanced Phonetic I guess it is working great to relieve the possibility of clots, for I have no sign of a clot returning. However, I am covered in bruises and my stomach is knots more, I feel bloated and uncomfortable. I am unable to drink even a small amount of coffee in the morning, even a few hours after having taken the dose. That being said, I have to take it for at least a year after my heart attack, so I guess I have to deal with these side effects. Review: Proton pump inhibitor and clopidogrel interaction - NeLM Find key information about medicines by name (prototype) The authors conclude �the current evidence does not justify the conclusion that PPIs decrease the clinical efficacy of clopidogrel. Nonetheless, until further reliable data become available, wide separation of PPI and clopidogrel dosing should in theory minimise any potential, though unproven, clinical interaction between these two widely used medications.�Clopidogrel: A Pharmacoeconomic Review of its Use in Patients with Non-ST Elevation Acute Coronary Syndromes [ Papers | Articles | Software | Books | Chapters | Authors | Institutions | Data (FRED) | JEL Classification | NEP reports | Search | New papers by email | Author registration | Rankings | Volunteers | FAQ | Blog | Plagiarism | Help! ] Clopidogrel (Plavix(R)) is a selective inhibitor of adenosine diphosphate-induced platelet aggregation. In patients with acute coronary syndromes (ACS) [unstable angina or non-ST-segment elevation myocardial infarction], clopidogrel plus aspirin (acetylsalicylic acid) for up to 1 year significantly reduced the risk of cardiovascular events relative to placebo plus aspirin in the well designed clinical trial CURE (Clopidogrel in Unstable angina to prevent Recurrent Events) and its substudy in patients undergoing percutaneous coronary intervention (PCI) [PCI-CURE].
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